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The human immunodeficiency virus (HIV) attacks the body’s immune system. A healthy immune system is what keeps you from getting sick.
Because HIV damages your immune system, you are more likely to get sick from bacteria and viruses. It is also harder for your body to fight off these infections when you do get them, so you may have trouble getter better. HIV is the condition that causes acquired immunodeficiency syndrome (AIDS).
HIV can only be passed from person to person through body fluids, such blood, semen and vaginal fluid. Children born to infected mothers can also become infected during pregnancy. The most common ways HIV is passed are:
By having unprotected anal, vaginal or oral sex with an infected person.
By sharing needles and syringes for injecting drugs with an infected person.
What happens after a person gets HIV?
After being infected with HIV, your body works hard to attack the virus. With your body fighting, the virus can’t make as many copies of itself. Even though you still have HIV, you’ll begin to look well and feel well again. The usual blood tests will be normal.
However, during this time, the virus is still attacking your lymph nodes. Lymph nodes are the centers of your body’s immune system. The virus may also attack your brain tissue and slowly cause damage there.
Over 10 to 15 years, HIV kills so many CD4 cells that your body can no longer fight off infections. When your CD4 cell count is 200 or less per mL, you have AIDS (a normal count is 600 to 1000). Once you have AIDS (which stands for acquired immunodeficiency syndrome), you can easily catch many serious infections.
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Since penicillin was introduced in the 1940s, scientists have developed more than 150 antibiotics to help stop the spread of infectious disease. But although these drugs have saved millions of lives, the misuse of antibiotics has caused problems. Their frequent use, often for conditions or infections that aren’t caused by bacteria, has given rise to bacteria that are resistant to many commonly used antibiotics.

Superbugs emerge when an antibiotic fails to kill all of the bacteria it targets, and the surviving bacteria become resistant to that particular drug and frequently other antibiotics as well. Doctors then prescribe a stronger antibiotic, but the bacteria quickly learn to withstand the more potent drug as well, perpetuating a cycle in which increasingly powerful drugs are required to treat infections.

Antibiotic-resistant strains of bacteria began to appear soon after penicillin was introduced. Today, antibiotic-resistant strains have become common, and bacteria resistant to a number of antibiotics have developed. Once only seen in hospitals, outbreaks of some resistant strains — such as methicillin-resistant Staphylococcus aureus (MRSA) — are appearing in the wider community.

For years, the potent antibiotic vancomycin (Vancocin) was a reliable last defense against certain severe infections, notably those caused by staphylococcus bacteria. But in recent years, some superbugs have become able to resist vancomycin.

While experts are working to develop new antibiotics and other treatments to keep pace with antibiotic-resistant strains of bacteria, infectious organisms adapt quickly. Antibiotic-resistant bacteria will continue to be a global health concern — and using antibiotics wisely is an important part of preventing their spread.

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A new research suggests that the weakened immune systems of people with HIV puts them at increased risk for at least seven types of cancer, but early diagnosis and treatment of HIV infection could help delay the onset of some of these cancers.
What is cancer? It’s a malignant tumor of potentially unlimited growth that expands locally by invasion and systemically by metastasis. And what is HIV? It’s any of several retroviruses and especially HIV-1 that infect and destroy helper T cells of the immune system causing the marked reduction in their numbers that is diagnostic of aids– called also aids virus, human immunodeficiency virus.
Researchers examined the incidence of three aids-defining cancers (Kaposi’s sarcoma, non-Hodgkin’s lymphoma and cervical cancer) and four non-aids-defining cancers (Hodgkin’s lymphoma, lung cancer, liver cancer and anal cancer) in 52,278 HIV-infected people.
The study authors also analyzed the association between immunodeficiency, viral load, antiretroviral treatment and the onset of the seven cancers. Overall, immunodeficiency increased the risk of all the cancers, and CD4 cell count was the most predictive risk factor for all the cancers except anal cancer. The cancer risk associated with viral load was lower than that associated with immunodeficiency, the researchers noted.
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